Correction: Short-term L-arabinose administration alleviates MASLD by remodeling gut microbiota and activating hepatic ATF5-dependent mitochondrial unfolded protein responseDownload PDF
Correction: Short-term L-arabinose administration alleviates MASLD by remodeling gut microbiota and activating hepatic ATF5-dependent mitochondrial unfolded protein response
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Heng Zhang1,2,3,4,#
1Endocrine and Metabolic Diseases Hospital of Shandong First Medical University, Shandong First Medical University & Shandong Academy of Medical Sciences, Jinan 250013, Shandong, China.
2Shandong Institute of Endocrine & Metabolic Diseases, Shandong First Medical University & Shandong Academy of Medical Sciences, Jinan 250013, Shandong, China.
3Shandong Provincial Key Medical and Health Laboratory of Translational Medicine in Endocrine and Metabolic Diseases, Jinan 250013, Shandong, China.
4Jinan Key Laboratory of Translational Medicine on Metabolic Diseases, Jinan 250013, Shandong, China.
5Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan 250355, Shandong, China.
6Department of Endocrinology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan 250021, Shandong, China.
7School of Basic Medicine, Shandong First Medical University and Shandong Academy of Medical Sciences, Jinan 250000, Shandong, China.
8Shandong University of Traditional Chinese Medicine, Jinan 250355, Shandong, China.
#These authors contributed equally to this work.
Heng Zhang1,2,3,4,#
,
Ruhan Chen1,2,3,4,#
1Endocrine and Metabolic Diseases Hospital of Shandong First Medical University, Shandong First Medical University & Shandong Academy of Medical Sciences, Jinan 250013, Shandong, China.
2Shandong Institute of Endocrine & Metabolic Diseases, Shandong First Medical University & Shandong Academy of Medical Sciences, Jinan 250013, Shandong, China.
3Shandong Provincial Key Medical and Health Laboratory of Translational Medicine in Endocrine and Metabolic Diseases, Jinan 250013, Shandong, China.
4Jinan Key Laboratory of Translational Medicine on Metabolic Diseases, Jinan 250013, Shandong, China.
5Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan 250355, Shandong, China.
6Department of Endocrinology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan 250021, Shandong, China.
7School of Basic Medicine, Shandong First Medical University and Shandong Academy of Medical Sciences, Jinan 250000, Shandong, China.
8Shandong University of Traditional Chinese Medicine, Jinan 250355, Shandong, China.
#These authors contributed equally to this work.
Ruhan Chen1,2,3,4,#
, ...
Xiaolei Wang1,2,3,4,8
1Endocrine and Metabolic Diseases Hospital of Shandong First Medical University, Shandong First Medical University & Shandong Academy of Medical Sciences, Jinan 250013, Shandong, China.
2Shandong Institute of Endocrine & Metabolic Diseases, Shandong First Medical University & Shandong Academy of Medical Sciences, Jinan 250013, Shandong, China.
3Shandong Provincial Key Medical and Health Laboratory of Translational Medicine in Endocrine and Metabolic Diseases, Jinan 250013, Shandong, China.
4Jinan Key Laboratory of Translational Medicine on Metabolic Diseases, Jinan 250013, Shandong, China.
5Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan 250355, Shandong, China.
6Department of Endocrinology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan 250021, Shandong, China.
7School of Basic Medicine, Shandong First Medical University and Shandong Academy of Medical Sciences, Jinan 250000, Shandong, China.
8Shandong University of Traditional Chinese Medicine, Jinan 250355, Shandong, China.
1Endocrine and Metabolic Diseases Hospital of Shandong First Medical University, Shandong First Medical University & Shandong Academy of Medical Sciences, Jinan 250013, Shandong, China.
2Shandong Institute of Endocrine & Metabolic Diseases, Shandong First Medical University & Shandong Academy of Medical Sciences, Jinan 250013, Shandong, China.
3Shandong Provincial Key Medical and Health Laboratory of Translational Medicine in Endocrine and Metabolic Diseases, Jinan 250013, Shandong, China.
4Jinan Key Laboratory of Translational Medicine on Metabolic Diseases, Jinan 250013, Shandong, China.
5Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan 250355, Shandong, China.
6Department of Endocrinology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan 250021, Shandong, China.
7School of Basic Medicine, Shandong First Medical University and Shandong Academy of Medical Sciences, Jinan 250000, Shandong, China.
8Shandong University of Traditional Chinese Medicine, Jinan 250355, Shandong, China.
#These authors contributed equally to this work.
Correspondence to: Dr. Dong Lin, School of Basic Medicine, Shandong First Medical University and Shandong Academy of Medical Sciences, Jinan 250000, Shandong, China. E-mail: lindong@sdfum.edu.cn; Dr. Xiaolei Wang, Endocrine and Metabolic Diseases Hospital of Shandong First Medical University, Shandong First Medical University & Shandong Academy of Medical Sciences, Jinan 250013, Shandong, China. E-mail: xiaoleiwang@sdfmu.edu.cn
Received: 5 Aug 2026 | Accepted: 11 Aug 2026 | Published: 14 Aug 2026
Academic Editor: Amedeo Lonardo | Copy Editor: Ting-Ting Hu | Production Editor: Ting-Ting Hu
Correction to Metab Target Organ Damage.2025;5:59.
In the original publication[1], the magnification indicated for the representative microscopic image in Figure 7C was incorrect. To ensure consistency with the original experimental records, the authors have replaced the published image with another representative field from the same experiment and the same sample, and corrected the magnification label accordingly.
Figure 7. The effect of ATF5 overexpression or knockdown on AML12 cells. (A) Validation of ATF5 knockdown cell model by qPCR; (B) Validation of ATF5 overexpression cell model by qPCR analysis; (C) Nile Red staining showing PA-induced lipid accumulation in AML12 cells; (D) Measurement of TG content in AML12 cells under different treatments; (E) qPCR analysis of the expression levels of ATF5, HSP60, and LONP1 in the ATF5 overexpression model; (F) TMRE staining relative fluorescence intensity. (****P < 0.0001, ***P < 0.001, **P < 0.01, and *P < 0.05). ATF5: Transcription factor 5; siATF5: small interfering RNA targeting transcription factor 5; OE-ATF5: transcription factor 5 overexpression; CON: control; PA: palmitic acid; AML12: alpha mouse liver 12; qPCR: TG: triglyceride; HSP60: heat shock protein 60; LONP1: Lon protease 1.
This correction is limited to the representative image and its magnification label. It does not affect the experimental design, quantitative analyses, statistical results, or data interpretation. Accordingly, the results and conclusions of the article remain unchanged.
1. Zhang H, Chen R, Zhang W, et al. Short-term L-arabinose administration alleviates MASLD by remodeling gut microbiota and activating hepatic ATF5-dependent mitochondrial unfolded protein response. Metab Target Organ Damage. 2025;5:59.
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Correction: Short-term L-arabinose administration alleviates MASLD by remodeling gut microbiota and activating hepatic ATF5-dependent mitochondrial unfolded protein response
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